How to Repair Damaged Skin Naturally

Regrow New Skin

This brand new method teaches you how to heal and regrow skin that was damaged in acute burn injuries, and grow the skin back better than it ever was before. This eBook was written as an alternative method to heal skin, as opposed to the traditional methods that have been used by doctors for years. This all new method uses recent discoveries and studies to show the best ways to get new skin in order to make brand new, smooth skin. Many customers have been really satisfied with the results that they got. Some people were able to get rid of scars, some people banished bedsores, some people were able to get rid of itches! No matter what sort of topical pain you are facing, from burns to acne to sores, you will be able to get rid of the pain and live comfortably and happily as a result!

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Contents: Ebook
Author: Ed Polaris
Price: $37.00

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Pathophysiology Of Cold Injuries

Heat is lost through the following four mechanisms radiation, convection, conduction, and evaporation. Radiation, in which heat is transferred by electromagnetic waves, accounts for almost two-thirds of heat loss in a cold environment. Radiant heat loss can be minimized by wearing appropriate warm clothing and minimizing the body surface area exposed to the cold environment. Conduction refers to the transfer of heat from warmer to cooler objects by direct contact. Heat loss through this mechanism is usually minimal, but it becomes a major source of heat loss in wet clothes or with cold-water immersion. Convection is the loss of heat to surrounding air and water vapor. Heat loss by convection is dependent on a combination of environmental factors, including wind velocity and temperature. The wind chill index, which estimates the equivalent temperature effect on exposed skin, is a combination of the ambient temperature and the wind velocity. It is a more important consideration than the...

Postherpetic Neuralgia and Other Neurologic Complications

Postherpetic Neuralgia

The acute herpes zoster radiculoneuritis affects mainly elderly patients with an incidence of 125 100,000 per year. The clinical onset of acute herpes zoster infection is heralded by pain in the affected segment (preherpetic neuralgia). The characteristic vesicopapular rash usually appears a few days after the onset of pain and takes 3-4 weeks to heal. In most patients, the rash and pain disappear completely within a period of 1-2 months. These patients develop neither local neuropathy nor other cutaneous sensory changes. In other patients, the acute neurocutaneous symptoms may be followed by irreversible skin damage and sensory abnormalities and, in a significant number of patients, there is persistent pain or the initial pain subsides and a second pain, often of different character, begins. This condition is called postherpetic neuralgia (PHN). In the overall population on average 12-20 suffer from pain at the time of skin healing and 2-5 at 1 year after zoster. The incidence of PHN...

Evolution and systematics

The tapeworms, together with the monogeneans and the trematodes, belong to a monophyletic taxon named Neoder-mata (i.e., having new skin ). This name reflects the fact that the ciliated epidermis of the larvae is replaced during the metamorphosis by a peculiar syncytial tissue (tegument or neodermis) occurring in adult worms. Main functions of the tegument are protective (against host's immune reactions and enzymes) and digestive (as a major site of absorption, metabolic transformations, and transport of nutrients). The tegument consists of a surface syncytial layer (distal cytoplasm) connected by cytoplasmic bridges with cell bodies (cytons). The cytons, containing nuclei and possessing powerful secretory apparatus, are situated deeply beneath the superficial muscle layers therefore, they are well protected against host's reactions. Their secretions permanently renovate the distal cytoplasm, which acts as a contact zone between the parasite body and the host's tissues and fluids....

Pathological Conditions

Bowen's disease (carcinoma in situ) Bowen's disease is a pre-invasive or in situ malignancy of the skin usually presenting as a red scaly patch. Most of these lesions present in a background of solar damage although it can occur in areas of non-sun-damaged skin where it may be associated with a higher incidence of internal malignancy. Bowen's disease is often treated by dermatologists with topical agents and may be biopsied to confirm the diagnosis and to exclude invasive malignancy. Occasionally there will be a biopsy to remove the lesion. Depending on whether the biopsy is excisional or diagnostic in intent the laboratory will receive either a curettage, shave, punch or elliptical specimen.


Clinical features of actinically damaged skin include coarseness, wrinkling, irregular pigmentation, telangiectasia, and scaliness, as well as a variety of premalignant and malignant neoplasms. The relative severity of these changes varies considerably among individuals. This undoubtedly reflects strong differences in past sun exposure and marked individual differences in vulnerabilities and repair capacities for solar insults. Photoaging usually involves most severely the face, neck, or extensor surface of the upper extremities 15 .


Based on studies that have been initiated by the group of Elias in San Francisco 25,26 , and taken further by others as well 8,27,28 , insight has been gathered into mechanisms and components involved in skin repair. Although the body of experiments in this direction was carried out on murine skin, evidence is accumulating that qualitatively similar mechanisms are operative in humans 29,30 . This leads to the view that BR products should have properties directed at re-establishing the broken skin barrier, which is accommodated by restoration of the physical integrity via application of missing basic components of the intracellular lipid matrix in combination with occlusive materials to stimulate repair mechanisms (Fig. 6). The function of the skin barrier is reflected by its ability to prevent excessive water loss. Consequently, transepidermal water loss (TEWL) is the parameter of choice to define the status of the skin barrier in this respect 31 . In this respect, criteria for BR...


Beta-carotene is known for its quenching activity on singlet oxygen. It would be an ideal partner for vitamin E and vitamin C to strengthen the antioxidant defense system of the skin. Oral supplementation with beta-carotene over several weeks has shown to reduce the risk of UV-induced skin damage 50 .

Vitamin E

Vitamin E is used in cosmetics for everyday use to strengthen the natural antioxidant potency of the skin and thus to better cope with oxidative stress. Most of the scientific background for the topical use of vitamin E stems from observations in context with UV light. Vitamin E is often used, therefore, in suncare products for improvement of the protection achieved with the sun filters. Even high SPF factors still allow the penetration of some UV light onto and into the skin. Whereas the sun filters absorb or reflect most of the rays on the surface of the skin, vitamin E acts on the inside and reduces the risk of damage that could be caused by rays passing through the sun filter barrier. Vitamin E helps, therefore, in the prevention of symptoms caused by UV-induced skin damage such as wrinkling and irregular pigmentation.


Individual differences that can predispose to the development of PTSD include age, history of emotional disturbance, social support, and proximity to the stressor. Eighty percent of young children who sustain a burn injury, for example, show symptoms of posttraumatic stress 1-2 years after the initial injury, but only 30 of adults who sustain this injury have symptoms after 1 year. Persons with a prior history of psychiatric treatment have a greater likelihood of developing the syndrome, presumable because the previous illness reflects a greater sensitivity to stress and persons with adequate social support are less likely to develop the disorder than persons with poor support.


Burns One study involving 27 patients with a partial-thickness burn injury found that topical aloe gel significantly increased the healing rate compared with controls who used a vaseline gauze. The mean healing time for the aloe gel group was 11.89 days compared with the control group, which was 18.18 days. Additionally, the aloe treatment brought about full epithelialisation after 14 days (Visuthikosol et al 1995).

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Tests in animal models Several animal studies support the application of aloe gel to skin damaged by frostbite as a means to maintain circulation and reduce the vasoconstrictive effects of thromboxane in the affected dermis (Heggers et al 1987, Klein & Penneys 1988, McCauley et al 1990, Miller & Koltai 1995). In combination with pentoxifylline, it will act synergistically to further increase tissue survival (Miller & Koltai 1995).

Burn Assessment

Superficial Partial-Thickness Burn Injury Superficial partial-thickness burn injury involves the papillary dermis, containing pain-sensitive nerve endings. Burn management, burns, burn blisters, or bullae may be present, and the burns usually appear pink and moist. These burn management, burns, and burn injuries heal with little or no scarring. Deep Partial-Thickness Burn Injury Deep partial-thickness burn injury damages both the papillary and reticular dermis. These injuries may not be burn management, burns, or burn painful, and often appear white or mottled pink. Deep partial-thickness burns can produce burn management, burns, and burn with significant scarring.


The gel reduces oxidation of arachidonic acid, thereby reducing PG synthesis and inflammation (Davis et al 1987). It inhibits the production of PGE2 by 30 and IL-8 by 20 , but has no effect on thromboxane B2 production in vitro (Langmead et al 2004). Following burn injury in vivo, A. vera was also found to inhibit inflammation by reducing leukocyte adhesion and decreasing the pro-inflammatory cytokines TNF-alpha and IL-6 (Duansak et al 2003).

Transmission risk

HIV is not a very contagious pathogen. The transmission rate after a high-risk contact is about 1 1000 to 1 100. Compared with HIV, the transmission rate for hepatitis C virus is 10 times higher, and 100 times higher for hepatitis B virus. Factors for the probability of transmission are the amount of incorporated virus and the exposure time. Contact with body fluids of a patient with a high viral load probably holds a higher risk of contagion than a similar contact with body fluids of a patient under HAART with a suppressed viral load. Additionally, quick removal of infectious material e.g. from damaged skin or mucosal membrane by washing or disinfection supposedly decreases the risk of an HIV infection. For percutaneous contact with HIV-containing blood, experts assume an infectiousness of 0.3 in total. According to retrospective data, calculations have been established to assess the transmission risks of accidental exposure more precisely (see Table 1). Exposition of inflammatory...

Dermatological Uses

Photo-aged skin Two double-blind studies investigating the effects of topical preparations of vitamin C on photo-damaged skin have demonstrated good results after 3 months' use (Fitzpatrick & Rostan 2002, Humbert et al 2003). One study tested a topical application of 5 vitamin C in a cream base, whereas the other used a newly formulated vitamin C complex having 10 ascorbic acid (water soluble) and 7 tetrahexyldecyl ascorbate (lipid soluble) in an anhydrous polysilicone gel base. Prevention of sunburn One controlled study found oral vitamin C (2000 mg day) in combination with vitamin E (1000 lU day) had a protective effect against sunburn after 8 days' treatment in human subjects (Eberlein-Konig et al 1998).