Amyloid Hypothesis And A Amyloidosis

The characteristic pathological signature of AD noted by Alzheimer is the presence of deposits of A/ peptide in extracellular plaques accompanied by tau deposits in intracellular tangles. The weight of the genetic evidence from familial forms of AD supports the A/ peptide as a prime mover in the etiology of the disease (Hardy and Selkoe, 2002). While it is clear that mutations in /APP can cause AD in the genetic cases, sporadic AD must involve other factors. If it is true that multimeric A/ is either an initiating factor and/or a chronic stressor, strategies to alleviate these effects should alter the course of the disease.

Unraveling Alzheimers Disease

Unraveling Alzheimers Disease

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