General Conclusion

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The high and abnormal activity of the HPA axis, low levels of sex steroids, and attenuating GH secretion in abdominal obesity suggests a central neuroendocrine dysregulation in abdominal obesity. Whether this is of primary importance for the evolution of abdominal obesity or merely a secondary phenomenon to the obese condition remains to be elucidated.

The finding that replacement with testosterone and GH to men with abdominal obesity are able to diminish the negative metabolic consequences of the visceral obesity suggest that the low levels of these hormones are of primary importance for the metabolic consequences associated with visceral/abdominal obesity.

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