Clinical observations

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The clinical effect of the above described relationship has to be analysed under six different aspects:

- The effect of Helicobacter pylori eradication on GORD symptoms and severity

- The effect of Helicobacter pylori infection and its eradication on proton pump inhibitor (PPI) efficacy

- The effect of Helicobacter pylori infection and its eradication on histology if long term PPI therapy is required for GORD maintenance therapy

- Helicobacter pylori infection and its role in Barrett's oesophagus

- The role of cagA-positive-strains in patients with Barrett's oesophagus

- The role of cagA-positive-strains in patients with oesophageal adenocarcinoma.

antrum predominant gastritis >

pangastritis

I Cytotoxinf t

\ Multifocal N^a trophic

Fig. 2. The pathophysiology of GORD (TLESR: transient lower esophageal sphincter relaxation; LES: lower esophageal sphincter pressure)

LOS pressure |

^ Cytotoxin t

^ Phospholipase f

Ammonia f

Fig. 2. The pathophysiology of GORD (TLESR: transient lower esophageal sphincter relaxation; LES: lower esophageal sphincter pressure)

Fig. 3. Impact of the pattern of gastritis: antrum predominant gastritis vs.pangastritis

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