Suggested Readings

1. Wigle ED. Novel insights into the clinical manifestations and treatment of hypertrophic cardiomyopathy. Curr Opin Cardiol 1995; 10:299-305.

2. Wigle ED, Rakowski H, Kimball BP, Williams WG. Hypertrophic cardiomyopathy: clinical spectrum and treatment. Circulation 1995; 92:1680-92.

3. Posma JL, van der Wall EE, Blanksma PK, van der Wall E; Lie KI. New diagnostic options in hypertrophic cardiomyopathy. Am Heart J. 1996; 132:1031-41.

4. Lakkis N. New treatment methods for patients with hypertrophic obstructive cardiomyopathy. Curr Opin Cardiol. 2000; 15:172-7.

5. Basso C, Corrado D, Thiene G. Cardiovascular causes of sudden death in young individuals including athletes. Cardiol Rev. 1999; 7:127-35.

6. Maron BJ. Hypertrophic cardiomyopathy. Lancet. 1997; 350:127-33.

7. Davies MJ, McKenna WJ. Hypertrophic cardiomyopathy--pathology and pathogenesis. Histopathology. 1995; 26:493-500.

8. Tardiff JC, Factor SM, Tompkins BD, Hewett TE, Palmer BM, Moore RL, Schwartz S, Robbins J, Leinwand LA. A truncated cardiac troponin T molecule in transgenic mice suggests multiple cellular mechanisms for familial hypertrophic cardiomyopathy. J Clin Invest 1998; 101:2800-11.

9. Gollob MH, Green MS, Tang ASL, Gollob T, Karibe A, Hassan A, Ahmad F, Lozado R, Shah G, Fananapazir L, Bachinski LL, Roberts R. Identification of a gene responsible for familial Wolff-Parkinson-White syndrome. N Engl J Med 2001; 344:1823-31.

Figure 14. Hypertrophic cardiomyopathy. The heart shows severe generalized left ventricula hypertrophy and asymmetric septal hypertrophy with a reduced ventricular cavity
Figure 15. Hypertrophic cardiomyopathy, left ventricle, histologic section. Thickened and sclerotic muscular artery. There is significant perivascular fibrosis (Hematoxylin and Eosin, 40X).
Figure 16. Hypertrophic cardiomyopathy. Haphazardly-arranged myocardial fibers (whorls) and interstitial scarring (Hematoxylin and Eosin, 40X)
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